ECG Made Easy Part 2 ECG Quiz

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ECG made easyPart 2 – ECG Quiz Presented by: Dr Randall Hendriks, Interventional Cardiologist – Western Australia1

? Axis1. Left2. Right3. Indeterminate

? Axis1. Left2. Right3. Indeterminate

? Axis1. Left2. Right3. IndeterminatePage 4

? Axis1. Left2. Right3. IndeterminatePage 5

? Axis1. Left2. Right3. IndeterminatePage 6

? Axis1. Left2. Right3. IndeterminateStandard limb lead reversal!Page 7

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 8

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 9

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 10

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 11

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 12

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 13

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 14

Tachycardia1.2.3.4.5.Atrial fibrillationAtrial flutterSVTSinus tachycardiaJunctionalPage 15

Tachycardia1.2.3.4.5.Atrial flutterAtrial fibrillationSVTSinus tachycardiaJunctionalPage 16

Tachycardia1.2.3.4.5.Atrial flutterAtrial fibrillationSVTSinus tachycardiaJunctionalPage 17

Bradycardia1. Sinus bradycardia2. First degree AV block3. Mobitz Type I(Wenckebach)4. Mobitz Type 25. Complete heart blockPage 18

Bradycardia1. Sinus bradycardia2. First degree AV block3. Mobitz Type I(Wenckebach)4. Mobitz Type 25. Complete heart blockPage 19

ST elevation e 20

ST elevation e 21

ST elevation e 22

ST elevation e 23

ST elevation e 24

ST elevation e 25

ST elevation – 30 year old Thai man with syncope1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 26

ST elevation – 30 year old Thai man with syncope 1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 27

ST elevation – young adult patient with pleuritic chest pain1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 28

ST elevation – young adult patient with pleuritic chest pain1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 29

Palpitations1. VT2. SVT with aberrancyPage 30

ST elevation – young adult patient with pleuritic chest pain1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 31

ST elevation – young adult patient with pleuritic chest pain1.2.3.4.5.Anterior MINormal repolarisationLV aneurysmPericarditisBrugada syndromePage 32

Palpitations1. Absence of RS?2. No – next questionPage 33

Palpitations1. R/S interval 100msec?2. Yes - VTPage 34

Can’t miss-life threatening ECGs35

Bradycardia and hypotensive in EDPage 36

Bradycardia and hypotensive in EDComplete heart blockPage 37

Wolff-Parkinson-White syndrome

Palpitations: 20 year old female with presyncopePage 40

Palpitations: 20 year old female with presyncopePre-excited AFPage 41

HypokalaemiaPage 42

Broad complex tachycardia – diagnosis?Page 43

Broad complex tachycardia – diagnosis?Torsades de Pointes – QT prolongationPage 44

Hypokalaemia Decreased extracellular K – hyperexcitability: re-entrantarrhythmias Increase amplitude and width of P wave Prolonged PR interval T flattening / inversion ST depression Prominent U waves Apparent long QT (QTU fusion) SVEs, VPBs SVT (AF, atrial flutter, atrial tachy) VT, VF and Torsades de PointesPage 45

HyperkalaemiaPage 46

Hyperkalaemia Increased extracellular K – reduces myocardial excitability Repolarisation changes:Peaked T waves Atrial paralysis:P wave widens and flattensPR prolongationP wave disappears Conduction abnormality and bradycardia:QRS prolongs / bizarreHigh grade AV block, junctional and ventricular escape rhythmsSinus brady or slow AFSine wave (pre terminal) Cardiac arrest:AsystoleVFPEA with bizarre wide QRSPage 47

HyperkalaemiaPage 48

Digoxin toxicity – PAT with block and VPBsPage 49

Bidirectional VT – digoxin toxicityPage 50

Digoxin toxicity Increased automaticity / decreased AV conduction:SVT with slow ventricular responsePVC’s, sinus brady, AFAny type of AV blockRegularised AF (AF with CHB)VT (polymorphic or bidirectional)Page 51

Intracranial haemorrhagePage 52

Intracranial haemorrhage Widespread giant T inversionQT prolongationBradycardia (Cushing reflex – brain stem herniation)ST elevation or depressionIncreased U wave amplitudeST, junctional, VPBs, AF(? Hypothalamic stimulation / autonomic dysregulation)Page 53

Massive pulmonary embolismPage 54

Massive pulmonary embolism Sinus tachycardia Complete or incomplete RBBB RV strainT inversion V1-4, II,III,aVF Right axis deviation Dominant R V1 P pulmonale SI, QIII, TIII in 20% only Clockwise rotation Atrial arrhythmias Non specific ST/T changesPage 55

Pacemaker malfunctionPage 56

70 year old, chest pain and diaphoresisPage 57

70 year old, chest pain and diaphoresisExtensive anterior MI (‘tombstoning” pattern)Page 58

Ischaemic sounding chest painPage 59

Ischaemic sounding chest painWellens’ Syndrome – proximal LAD stenosisPage 60

LMCA occlusionPage 61

LMCA occlusion Widespread ST depression (leads I, II, V4 – 6) ST elevation aVR 1mm ST elevation aVR V1 Can also see in:prox LAD occlusionsevere triple vessel diseasediffuse subendocardial ischaemia (ie. post resuscitation) aVR records electrical activity right upper portion of heart,including RVOT and basal IV septumPage 62

De Winter’s T waveAnterior STEMI equivalentST depression and peaked T waves inprecordial leadsSeen in 2% acute LAD occlusionsYounger / male / hypercholesterolaemiaCode STEMIPage 64

Middle aged female presents with dyspnoea, prior mastectomy for breastcancer.Page 65

Middle aged female presents with dyspnoea, prior mastectomy for breastcancer.QRS alternans – pericardial effusionPage 66

Ventricular flutterPage 67

Ventricular flutter Continuous monomorphic sine wave No identifiable P, QRS or T wave Rate 200 Extreme form of VT Rapid degeneration into VFPage68

HypothermiaBradyarrhythmias (any)Osborn waves ( J waves positive deflection at J point)Prolonged PR, QRS and QTShivering artefactVPBsCardiac arrest due to VT, VF orasystolePage 69

HypothyroidismBradycardiaLow QRS voltageWidespread T wave inversionQT prolongationFirst degree AV blockIVCDsMyxoedematous deposits in myocardiumDecreased SNS activityLess thyroxine – decreased inotropy /chronotropyPage 70

The End71

ECG made easy Part 2 –ECG Quiz 1 Presented by: Dr Randall

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